Module 8 Active Learning Guide

Module 8 Active Learning Guide

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Instructions:  Complete the module active learning guide as you work through the module content. You will submit this for points.

Reading Focus Areas

  1. When conducting all of your readings this week, keep an active Know, What to know, and what you Learned (KWL) chart.
Know Want to Know What you Learned
     
     
     
     
     
     
     

 

  1. Define 10 terms from your chapter readings

 

Term Definition
   
   
   
   
   
   
   
   
   
   

 

Deeper Level Questions

 

  1. Make a list of causes and treatments for the patient with DKA. Include lab monitoring, anion gap calculation and guidelines, and transition to sub q insulin.

 

 

 

 

  1. Develop a continuum chart with the signs and symptoms for myxedema coma and thyroid crisis. Include treatments for both.

 

 

 

 

  1. Review the case study for the patient with pancreatitis. Using the SBAR format, develop a paragraph to report to the physician or practitioner. **Note** Mr. Harrison’s pain is not controlled with Morphine.

 

  1. Joe Harrison, a 62-year-old bank executive, presents to the emergency room with severe abdominal pain. He describes the pain as excruciating, and indicates it is located in the midepigastrium with radiation into his back. The patient states he has not eaten anything in the past 24 hours, but two days ago attended a wedding dinner and consumed a large meal and about 4 – 5 alcoholic beverages. Mr. Harrison admits to being a “social drinker,” ingesting 2 – 3 alcoholic beverages several days a week. Based upon his clinical presentation and history, Mr. Harrison is admitted with a diagnosis of Rule-out Pancreatitis.

 

  1. Blood and urine samples are sent to the lab, and the results indicate that the serum amylase and lipase are markedly elevated. How do these findings correlate to the diagnosis of acute pancreatitis?

 

 

  1. The nurse notes that Mr. Harrison’s stool is pale and bulky, while his urine is a dark tea color. Correlate these findings to the pathophysiology of acute pancreatitis.

 

 

  • Mr. Harrison’s WBC is 18,500, and his serum glucose is 325. His LDH is 300 IU/L and the AST is 120 U/ml. Based upon the Criteria for Predicting the Severity of Pancreatitis, what is Mr. Harrison’s mortality risk?

 

 

  1. Mr. Harrison develops hypocalcemia secondary to the acute pancreatitis. What nursing interventions should the nurse implement related to this complication?